Our DNA contains approximately 30,000 genes. Within our cells, our DNA is organized into 23 pairs of chromosomes. Genetic variations in our DNA are called SNPs (Single Nucleotide Polymorphisms). If we compare the DNA of two individuals, they will appear to be over 99% identical, (the DNA of identical twins is nearly 100% identical). However, the crucial variations that occur within the last 1% are a factor in making us genetically unique. Genetic analysis and research has determined that many identified SNPs can be associated with a predictable increase in risk of certain conditions commonly associated with aging. Genetic research has also revealed that our genes can be activated in a positive or negative fashion based upon the quality of the environment in which they operate.

How is our cellular health damaged over time?

The Unified DNA Damage Theory of Aging, proposed by Vincent C. Giampapa, M.D., F.A.C.S., explains the effects of many factors on our genetic health. As the theory explains, there are five key cellular processes that can be a factor in the aging process. These can be broadly described as:

	Methylation – The turning on and off of specific genes, generally related to heart and vascular health
	Inflammation – Inflammatory molecules present in cells
	Glycation – Blood sugar effects on proteins and body fat
	Oxidation – The production of free radicals
	DNA Repair – The ongoing regeneration of our cellular structures

Each one of these processes is controlled by a specific group of genes. If these cellular processes become poorly controlled over time or were sub-optimal from poor genetic inheritance, we may gradually come to suffer conditions that can be associated with a decline in overall cellular health.